Title of article
A Novel and De Novo Spontaneous Point Mutation (Glu271 STOP) of the Antithrombin Gene Results in a Type I Deficiency and Thrombophilia
Author/Authors
Waye، John S. نويسنده , , Tarantino، Michael D. نويسنده , , Curtis، Sharon M. نويسنده , , Johnson، G. Stephan نويسنده , , Blajchman، Morris A. نويسنده ,
Issue Information
روزنامه با شماره پیاپی سال 1999
Pages
-125
From page
126
To page
0
Abstract
A study of the properties of the neutron production target for the China accelerator-driven system induced from 150-MeV incident proton energy and a 3-mA beam current is performed. The selection of the material and geometry design for the target; the physics properties of the target involved in this work including the neutron yield, energy, and space distribution for the neutrons leaked from the target; and the spallation fragment of charge and mass distribution have been investigated using the SHIELD code. An analysis of the target radiation damage and energy deposit is also carried out.
Keywords
antithrombin , antithrombin deficiency , Thrombophilia , type I AT deficiency
Journal title
American Journal of Hematology
Serial Year
1999
Journal title
American Journal of Hematology
Record number
25601
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