Title of article :
Genome Editing of Isogenic Human Induced Pluripotent Stem Cells Recapitulates Long QT Phenotype for Drug Testing
Author/Authors :
Wang، نويسنده , , Yongming and Liang، نويسنده , , Wei-Ping and Lan، نويسنده , , Feng and Wu، نويسنده , , Haodi and Lisowski، نويسنده , , Leszek and Gu، نويسنده , , Mingxia and Hu، نويسنده , , Shijun and Kay، نويسنده , , Mark A. and Urnov، نويسنده , , Fyodor D. and Shinnawi، نويسنده , , Rami and Gold، نويسنده , , Joseph D. and Gepstein، نويسنده , , Lior and Wu، نويسنده , , Joseph C.، نويسنده ,
Issue Information :
روزنامه با شماره پیاپی سال 2014
Pages :
9
From page :
451
To page :
459
Abstract :
AbstractBackground induced pluripotent stem cells (iPSCs) play an important role in disease modeling and drug testing. However, the current methods are time-consuming and lack an isogenic control. ives tudy sought to establish an efficient technology to generate human PSC-based disease models with isogenic control. s n channel genes KCNQ1 and KCNH2 with dominant negative mutations causing long QT syndrome types 1 and 2, respectively, were stably integrated into a safe harbor AAVS1 locus using zinc finger nuclease technology. s clamp recording revealed that the edited iPSC-derived cardiomyocytes (iPSC-CMs) displayed characteristic long QT syndrome phenotype and significant prolongation of the action potential duration compared with the unedited control cells. Finally, addition of nifedipine (L-type calcium channel blocker) or pinacidil (KATP-channel opener) shortened the action potential duration of iPSC-CMs, confirming the validity of isogenic iPSC lines for drug testing in the future. sions udy demonstrates that iPSC-CM-based disease models can be rapidly generated by overexpression of dominant negative gene mutants.
Keywords :
disease models , Drug testing , Genome editing , long QT syndrome , stem cells
Journal title :
JACC (Journal of the American College of Cardiology)
Serial Year :
2014
Journal title :
JACC (Journal of the American College of Cardiology)
Record number :
1759005
Link To Document :
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