Author/Authors :
Al-Shaer، نويسنده , , Moutasim H. and Raghuveer، نويسنده , , Geetha and Browning، نويسنده , , Roger and Sinkey، نويسنده , , Christine A. and Chenard، نويسنده , , Catherine and Stumbo، نويسنده , , Phyllis and Haynes، نويسنده , , William G.، نويسنده ,
Abstract :
Homocysteine may contribute to systolic hypertension and cardiac events by decreasing conduit artery compliance and inducing endothelial dysfunction. The effects of the experimental induction of hyperhomocysteinemia on systemic arterial compliance and pulsewave velocity are unclear, with contradictory results from previous studies. The investigators tested whether oral methionine impairs brachial artery compliance in addition to endothelial function.
