Title of article :
Molecular cloning, characterization and expression analysis of three heat shock responsive genes from Haliotis diversicolor
Author/Authors :
Huang، نويسنده , , Yitao and Cai، نويسنده , , Xiuhong and Zou، نويسنده , , Zhihua and Wang، نويسنده , , Shuhong and Wang، نويسنده , , Guodong and Wang، نويسنده , , Yilei and Zhang، نويسنده , , Ziping، نويسنده ,
Issue Information :
روزنامه با شماره پیاپی سال 2014
Pages :
10
From page :
590
To page :
599
Abstract :
In this study, molecular characterization and expression of three heat shock responsive genes were analyzed as indicators to understand the mechanism of heat shock response of small abalone Haliotis diversicolor under stresses. The full length cDNA of heat shock transcriptional factor 1 (HdHSF1), heat shock factor binding protein 1(HSBP1), and heat shock protein 90 (HdHSP90) are 1548 bp, 809 bp, and 2592 bp respectively, encoding a protein of 515 aa, 75 aa, and 728 aa respectively. Real time quantitative PCR analysis revealed that these three genes are constitutively expressed in 7 selected tissues. The expression level of HdHSF1 in gills was higher than that in other tissues (p < 0.05). The highest expression level of HdHSBP1 was detected in hemocytes. The highest expression level of HdHSP90 was in the digestive tract and colleterial gland. The HdHSF1 expression level in the gills was up-regulated significantly (p < 0.05) after thermal stress and hypoxia exposure respectively. On the contrary, HdHSBP1 was down-regulated both in gills and hemocytes after thermal stress and the same as in gills after hypoxia stress. HdHSP90 expression level was also up-regulated in gills and hemocytes after both thermal and hypoxia stresses. These results indicated that these three heat shock responsive genes play important roles in response to thermal and hypoxia stress.
Keywords :
HSP90 , STRESS , Haliotis diversicolor , HSBP1 , HSF1
Journal title :
Fish and Shellfish Immunology
Serial Year :
2014
Journal title :
Fish and Shellfish Immunology
Record number :
2112962
Link To Document :
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