Author/Authors :
Del Carpio-Orantes, Luis Department of Internal Medicine - Instituto Mexicano del Seguro Social, Veracruz, Mexico
Abstract :
After the arrival of Zika to the Americas in 2014, and its rapid spread across the continent in less than two years, colonizing from the southern cone to North America and conditioning well-established neurological and teratogenic syndromes such as Guillain Barre syndrome and Zika congenital syndrome (characterized by microcephaly, arthrogryposis, and other congenital disorders), which had their origin in regions of Asia and Oceania, where unlike their behavior in these lands, they were conferred benignity and zero mortality. However, when presenting mutations in American lands, Zika had greater virulence and pathogenicity that led to new manifestations not observed in Asian regions such as full neurotropic and teratogenic potential, which has led to a new American lineage (1, 2). Luckily, like any pandemic, Zika has diminished its potential to become a subclinical virus. However, there are still few case reports that have occurred in the same way with dengue and chikungunya.