Author/Authors :
Ashkani Esfahani, Soheil shiraz university of medical sciences - Student Research Committee, ايران , Esmaeilzadeh, Elmira shiraz university of medical sciences - Student Research Committee, ايران , Bagheri, Fereshte shiraz university of medical sciences - Student Research Committee, ايران , Emami, Yasaman shiraz university of medical sciences - Student Research Committee, ايران , Farjam, Mojtaba fasa university of medical sciences - Department of Pharmacology, ايران
Abstract :
Liver plays the role of detoxifier and excreter of destructive agents against body intoxication. Liver injury occurs following by histopathological changes including degeneration, necrosis, and atrophy of liver parenchymal cells with interstitial connective tissue as well as increasing in liver enzymes, such as aspartate aminotransferase/alanine aminotransferase (AST/ALT), alkaline phosphatase (ALP), and plasma total bilirubin level (TBili) (1). Acute Hepatic Encephalopathy (AHE) is described as a neuropsychiatric syndrome which can be pursued by urea cycle defect as well as acute (ALF) and chronic liver failure (CLF).
