Title of article :
Left ventricular outflow tract obstruction due to residual native valve following mitral valve replacement
Author/Authors :
Shipman, Justin Department of Internal Medicine - Mayo Clinic - Arizona - United States , Agasthi, Pradyumna Department of and Cardiovascular Medicine - Mayo Clinic - Arizona - United States , DeValeria, Patrick Department of Cardiovascular Surgery - Mayo Clinic - Arizona-United States , Mookadam, Farouk Department of Internal Medicine - Mayo Clinic - Arizona - United States , Arsanjani, Reza Department of Internal Medicine - Mayo Clinic - Arizona - United States
Abstract :
An 84-year-old female with history of bioprosthetic mitral
valve replacement four years earlier presented with a progressively worsening dyspnea on exertion. A transthoracic echocardiogram (TTE) showed a mean gradient of 13 mmHg across the
bioprosthetic valve (Fig. 1a). The TTE also noted a left ventricular
outflow tract obstruction (LVOTO) gradient due to residual native
valve tissue (peak gradient >130 mm Hg) (Fig. 1b, arrow) and an
estimated right ventricular systolic pressure of 70 mm Hg. The
transesophageal echocardiogram (TEE) corroborated that two
out of three leaflets on the bioprosthetic valve had a significantly
reduced motion (Fig. 1c and Video 1). In addition, a significant
systolic anterior motion of the native mitral valve anterior leaflet (red arrows) was observed which had not been resected
throughout the original surgery, resulting in a significant dynamic
LVOTO (Fig. 1d and 1e and Video 1). She subsequently underwent
redo bioprosthetic mitral valve replacement and resection of
the native anterior mitral valve leaflet (Fig. 1f). The resected bioprosthetic valve revealed findings consistent with a degenerated
valve prosthesis with calcified leaflets and significantly restricted motion (Fig. 1g, white arrows). Her postoperative course was
unremarkable, and she was discharged on postoperative day 7.
Postoperative LVOTO may occur for a variety of reasons, including abnormal prosthetic position, hypercontractile ventricle,
left ventricular hypertrophy, and a small ventricular cavity (1-3).
Dynamic obstruction secondary to the preservation of native
anterior mitral valve leaflet has also been outlined (our patient)
(4, 5). This problem was likely exacerbated by the presence of a
prosthetic stenosis. This case also highlights the importance of
intraoperative TEE.
Keywords :
Left ventricular outflow tract obstruction , mitral valve replacement , systolic anterior motion
Journal title :
The Anatolian Journal of Cardiology: Andolu Kardiyoloji Dergisi