Author/Authors :
Sayan, Hatice Kayseri Education and Training Hospital - Department of Physical Medicine and Rehabilitation, Turkey , Çalış, Havva Talay Kayseri Education and Training Hospital - Department of Physical Medicine and Rehabilitation, Turkey , Sütbeyaz, Serap Tomruk Kayseri Education and Training Hospital - Department of Physical Medicine and Rehabilitation, Turkey
Abstract :
Complex regional pain syndrome (CRPS) is characterized by vasomotor and sudomotor changes. Its etiology generally includes trauma, fracture, stroke, and coronary disease. It is evaluated in two subtypes: CRPS type I (reflex sympathetic dystrophy), presenting with complex regional pain syndromes accompanied by spontaneous pain, hyperalgesia, allodynia, edema, autonomic anomalies, and trophic changes; regional pain occurring after minor injuries and fractures in the extremities; and coexisting vasomotor, sudomotor changes, and sensorial changes, and CRPS type II (coxalgia), including major peripheral nerve injuries, in addition to all of these symptoms (1, 2). Although the pathogenesis of CRPS is not exactly known, peripheral and central sensitization, which causes neurogenic inflammation, is held responsible.
