Title of article :
An uncommon presentation of hepatic hydatid cyst leading to biliary cirrhosis and portal hypertension
Author/Authors :
Asadzadeh-Aghdaei, Hamid Gastroenterology and Liver Diseases Research Center - Research Institute for Gastroenterology and Liver Diseases - Shahid Beheshti University of Medical Sciences - Tehran, Iran , Moshari, Mohammad Reza Taleghani Hospital - Shahid Beheshti University of Medical Sciences - Tehran, Iran , Zandi, Reza Taleghani Hospital - Shahid Beheshti University of Medical Sciences - Tehran, Iran , Karimi, Mohammad Ali Gastroenterology and Liver Diseases Research Center - Research Institute for Gastroenterology and Liver Diseases - Shahid Beheshti University of Medical Sciences - Tehran, Iran , Salari, Sina Taleghani Hospital - Shahid Beheshti University of Medical Sciences - Tehran, Iran , Ketabi Moghadam, Pardis No Affiliation
Pages :
4
From page :
145
To page :
148
Abstract :
Hydatid disease is an ongoing issue in endemic areas. Hydatid cysts can be seen in any organ but, liver is one of the most common involved organs. Cystobiliary communication as an overwhelming complication of hepatic hydatid cysts can contribute to the obstructive jaundice, cholangitis, sepsis and even biliary cirrhosis if left untreated. The patient we are trying to present is a 61-yearold farmer who presented with obstructive jaundice, multiple common bile duct stones and biliary cirrhosis attributed to a long-lasting untreated hepatic hydatid cyst. Portal hypertension is introduced to be an uncommon presentation of hydatid cyst. Extrinsic compression of the porta hepatis and obstruction of inferior vena cava are amongst major causes of hydatidosis leading up to portal hypertension as reported in the literature. Portal hypertension in the presented case is proposed to emerge from long-lasting cystobiliary communication ending in biliary cirrhosis.
Keywords :
Biliary cirrhosis , Hydatid cyst , Hepatic
Journal title :
Gastroenterology and Hepatology From Bed to Bench
Serial Year :
2020
Record number :
2659365
Link To Document :
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