Author/Authors :
Kammoun, Khawla Hedi Chaker Hospital - Department of Nephrology, Tunisia , Jarraya, Faiçal Hedi Chaker Hospital - Department of Nephrology, Tunisia , Ben Hmida, Mohamed Hedi Chaker Hospital - Department of Nephrology, Tunisia , Khebir, Abedelmajid Habib Bourguiba Hospital - Department of Histopathology, Tunisia , Kharrat, Mahmoud Hedi Chaker Hospital - Department of Nephrology, Tunisia , Boudawara, Tahia Habib Bourguiba Hospital - Department of Histopathology, Tunisia , Mnif, Jamel Habib Bourguiba Hospital - Department of Radiology, Tunisia , Hachicha, Jamil Hedi Chaker Hospital - Department of Nephrology, Tunisia
Abstract :
Tumoral calcinosis and calciphylaxis are uncommon but severe complications in uremic patients. They occur generally after long-term hemodialysis (HD) treatment explained by advanced secondary hyperparathyroidism and longstanding high calcium phosphorus product (Ca × P). Other factors such granulomatous diseases may worsen the calcium phosphate homeostasis alterations. We report a young male patient treated by HD for 6 years who developed tuberculosis in addition to tumoral calcinosis and calciphylaxis.
