Title of article
Biochemical but not clinical vitamin A deficiency results from mutations in the gene for retinol binding protein
Author/Authors
Biesalski، Hans K نويسنده , , Frank، Jurgen نويسنده , , Beck، Susanne C نويسنده , , Heinrich، Felix نويسنده , , Illek، Beate نويسنده , , Reifen، Ram نويسنده , , Gollnick، Harald نويسنده , , Seeliger، Mathias W نويسنده , , Wissinger، Bemd نويسنده , , Zrenner، Eberhart نويسنده ,
Issue Information
روزنامه با شماره پیاپی سال 1999
Pages
-930
From page
931
To page
0
Abstract
The 236U(nth, f) cross section was measured for the first time at the high-flux reactor of the Institut LaueLangevin in Grenoble, yielding a value of (0.3 ± 1.0) mb. This means that f 1.3 mb, which is about two orders of magnitude lower than previously adopted. This result was obtained by combining a highly enriched 236U sample with a very clean neutron beam and assuming that the Westcott factor gf = 1 for 236U (n, f) with cold neutrons. The new value is compatible with the latest fission resonance data and with the subthreshold character of the thermal neutron induced fission of 236U.
Keywords
mutation , vitamin A deficiency , retinol , Transthyretin , night blindness , retinyl esters , genomic sequence analysis , Retinol binding protein
Journal title
American Journal of Clinical Nutrition
Serial Year
1999
Journal title
American Journal of Clinical Nutrition
Record number
37913
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