Title of article :
Lesion progression and plaque composition are not altered in older apoE−/− mice lacking tumor necrosis factor-α receptor p55
Author/Authors :
Erwin Blessing، نويسنده , , Florian Bea، نويسنده , , Cho-chou Kuo، نويسنده , , Lee Ann Campbell، نويسنده , , Brian Chesebro، نويسنده , , Michael E. Rosenfeld، نويسنده ,
Issue Information :
روزنامه با شماره پیاپی سال 2004
Abstract :
Inflammatory processes are an integral component of the initiation, progression, and destabilization of atherosclerotic lesions. Tumor necrosis factor-α (TNF-α) is considered a primary mediator of inflammatory processes.
Keywords :
Tumor necrosis factor-? , Plaque rupture , inflammation , hyperlipidemia , atherosclerosis
Journal title :
Atherosclerosis
Journal title :
Atherosclerosis
