DocumentCode :
2205841
Title :
Calcium signaling of chondrocytes under osmotic stress and mechanical stimulation
Author :
Li, Wen ; Park, Miri ; Kirn-Safran, Catherine ; Wang, Liyun ; Lu, X. Lucas
Author_Institution :
Univ. of Delaware, Newark, DE, USA
fYear :
2012
fDate :
16-18 March 2012
Firstpage :
223
Lastpage :
224
Abstract :
Chondrocytes play a critical role in cartilage remodeling by mediating the biosynthesis, organization, and modification of extracellular matrix (ECM). They are highly sensitive to the surrounding mechanical and osmotic environments. One of the earliest responses of chondrocytes to stimuli is a transient oscillation in intracellular Ca2+ concentration ([Ca2+]i). The major objective of this study was to investigate and compare the Ca2+ signaling of chondrocytes, including both primary cells and chondrogenic cell line, under mechanical stimulus and osmotic stress. The roles of seven essential pathways in Ca2+ signaling were further examined. Cells were stained with calcium indicator and monitored under confocal microscopy. The results suggested that i) the two types of cells have different calcium signaling mechanisms; ii) mechanical stimulation and osmotic stress employ different mechanisms to initiate calcium responses; iii) osmotic stress could be the major regulation mechanism on the metabolisms of chondrocytes in daily activities.
Keywords :
biochemistry; biomechanics; biomembrane transport; bone; calcium; optical microscopy; osmosis; Ca; biosynthesis; calcium signaling; cartilage remodeling; chondrocytes; chondrogenic cell line; confocal microscopy; extracellular matrix; mechanical stimulation; osmotic stress; transient oscillation; Calcium; Electronic countermeasures; Erbium; Extracellular; Imaging; Stress; Transient analysis;
fLanguage :
English
Publisher :
ieee
Conference_Titel :
Bioengineering Conference (NEBEC), 2012 38th Annual Northeast
Conference_Location :
Philadelphia, PA
ISSN :
2160-7001
Print_ISBN :
978-1-4673-1141-0
Type :
conf
DOI :
10.1109/NEBC.2012.6207044
Filename :
6207044
Link To Document :
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