DocumentCode
3318987
Title
Notice of Retraction
A Model of Mechanisms of Cellular Defending Genome Stress Based on KTAP Framework
Author
Jinpeng Qi ; Yongsheng Ding ; Yizhi Wu ; Ying Zhu
Author_Institution
Coll. of Inf. Sci. & Technol., Donghua Univ., Shanghai, China
fYear
2011
fDate
10-12 May 2011
Firstpage
1
Lastpage
4
Abstract
Notice of Retraction
After careful and considered review of the content of this paper by a duly constituted expert committee, this paper has been found to be in violation of IEEE´s Publication Principles.
We hereby retract the content of this paper. Reasonable effort should be made to remove all past references to this paper.
The presenting author of this paper has the option to appeal this decision by contacting TPII@ieee.org.
Under acute perturbations from outer environment, a mathematical model of cellular self-defense mechanisms is proposed by using the method of Kinetic Theory of Active Particles (KTAP). In the processes of DNA damage repair, DNA damage and Repair Protein (RP) generating, DSBC synthesizing are implemented by the particle interactions between the pairs of DNA and acute Ion Radiation (IR), DNA damage and RP. Furthermore, cellular self-repair capability, cellular activity and genome stability are analyzed under different IR dose domains.
After careful and considered review of the content of this paper by a duly constituted expert committee, this paper has been found to be in violation of IEEE´s Publication Principles.
We hereby retract the content of this paper. Reasonable effort should be made to remove all past references to this paper.
The presenting author of this paper has the option to appeal this decision by contacting TPII@ieee.org.
Under acute perturbations from outer environment, a mathematical model of cellular self-defense mechanisms is proposed by using the method of Kinetic Theory of Active Particles (KTAP). In the processes of DNA damage repair, DNA damage and Repair Protein (RP) generating, DSBC synthesizing are implemented by the particle interactions between the pairs of DNA and acute Ion Radiation (IR), DNA damage and RP. Furthermore, cellular self-repair capability, cellular activity and genome stability are analyzed under different IR dose domains.
Keywords
DNA; biological effects of ionising particles; cellular effects of radiation; genomics; kinetic theory; molecular biophysics; physiological models; proteins; DNA damage; DNA damage repair; KTAP framework; active particles; acute ion radiation; cellular activity; cellular defending genome stress; cellular self-defense mechanisms; cellular self-repair capability; genome stability; kinetic theory; repair protein; Bioinformatics; DNA; Genomics; Kinetic theory; Maintenance engineering; Mathematical model; Stability analysis;
fLanguage
English
Publisher
ieee
Conference_Titel
Bioinformatics and Biomedical Engineering, (iCBBE) 2011 5th International Conference on
Conference_Location
Wuhan
ISSN
2151-7614
Print_ISBN
978-1-4244-5088-6
Type
conf
DOI
10.1109/icbbe.2011.5780105
Filename
5780105
Link To Document